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Dissertation
Year: 1998 Publisher: Liège : Université de Liège. Faculté de médecine (ULg). Département de clinique et pathologie médicales,
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Dissertation
Year: 1999 Publisher: Liège : Université de Liège. Faculté de Médecine. Institut supérieur d'éducation physique,
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Dissertation
Year: 1998 Publisher: Liège : Université de Liège. Faculté de médecine (ULg). Département de clinique et pathologie médicales,
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Dissertation
Year: 1998 Publisher: Liège : Université de Liège. Faculté de médecine (ULg). Département de clinique et pathologie médicales,
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Book
Year: 2018 Publisher: Basel : MDPI - Multidisciplinary Digital Publishing Institute,
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Apoptosis is a form of programmed cell death that enables the removal of damaged, infected, or otherwise unwanted cells in a controlled manner. Apoptosis can be initiated by multiple independent pathways that ultimately converge at a point where proteolytic enzymes belonging to the caspase family are activated, which dismantle the apoptotic cell. Multicellular organism have employed apoptotic mechanisms during host defence in response to viral infection to limit or prevent viral spread and replication. Consequently, viruses have evolved sophisticated molecular countermeasures to disarm host apoptotic defences, and this series of reviews and primary research articles in this Special Issue explores the intricate molecular interplay between viruses and their hosts when they battle for control of host apoptotic check-points.
Book
Year: 1995 Publisher: Cold Spring Harbor (N.Y.) : Cold Spring Harbor laboratory,
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Book
Year: 2001 Publisher: Cold Spring Harbor (N.Y.) : Cold Spring Harbor laboratory,
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ISBN: 9783718654611 371865461X Year: 1993 Publisher: Chur: Harwood,
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Multi
ISBN: 9780128157633 0128157631 0128157623 9780128157626 Year: 2021 Publisher: London : Academic Press,
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Book
Year: 2018 Publisher: Basel : MDPI - Multidisciplinary Digital Publishing Institute,
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Apoptosis is a form of programmed cell death that enables the removal of damaged, infected, or otherwise unwanted cells in a controlled manner. Apoptosis can be initiated by multiple independent pathways that ultimately converge at a point where proteolytic enzymes belonging to the caspase family are activated, which dismantle the apoptotic cell. Multicellular organism have employed apoptotic mechanisms during host defence in response to viral infection to limit or prevent viral spread and replication. Consequently, viruses have evolved sophisticated molecular countermeasures to disarm host apoptotic defences, and this series of reviews and primary research articles in this Special Issue explores the intricate molecular interplay between viruses and their hosts when they battle for control of host apoptotic check-points.
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