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Mineralocorticoid Receptor Antagonists --- Blood Pressure --- Mineralocorticoid Receptor Antagonists --- Stress, Physiological --- pharmacology --- drug effects --- physiology

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It has been recognized that moderation in the use of salt (sodium chloride) prevents the tendency of blood pressure to increase with age. On the other hand, the abuse of salt frequently leads to increases in blood pressure and contributes to the development of hypertension, particularly in overweight or obese people, in people with diabetes, in the elderly, and in genetically predisposed subjects. This Special Issue aims to provide a better understanding of the relationship between sodium intake and related diseases, in particular: (i) the effect on health status and description of the biochemical processes involved; and (ii) the use of salt and related risks. The main topics are studies in the management and treatment of sodium-intake-related diseases, epidemiological studies of the relationship between salt intake and related diseases, focuses on the mechanism of action; delineation of the mechanisms of action, and in vitro and in vivo studies.

Research & information: general --- Biology, life sciences --- Food & society --- tight junction --- Na+ cotransport --- leaky epithelia --- blood pressure --- inflammation --- mineralocorticoid receptor --- Rac1 --- renal injury --- salt-sensitive hypertension --- salt intake --- sodium --- hypertension --- cardiovascular risk --- mortality --- prognosis --- salt --- heart failure --- ambulatory heart failure --- epidemiological studies --- MST3 --- STK24 --- high potassium --- ENaC --- NKCC2 --- SPAK --- OSR1 --- WNK4 --- n/a

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This book presents the latest research published in the Special Issue Preventing the Adverse Effects of SARS-CoV-2 Infection and COVID-19 through Diet, Supplements, and Lifestyle in the journal Nutrients. In this book, we explore the role of supplements, lifestyle choices, and nutritional status on COVID-19 patient outcomes and risk reduction.

Research & information: general --- Biology, life sciences --- Biochemistry --- vitamin D --- COVID-19 --- SARS-CoV-2 --- severity --- mortality --- United Arab Emirates --- clinical trial --- saudi --- vitamin D insufficiency --- glycyrrhizin --- mineralocorticoid receptor --- toll like receptor 4 --- angiotensin converting enzyme --- aldosterone --- coronavirus --- exercise --- emotional eating --- pandemic --- quarantine --- questionnaire --- sedentariness --- New York --- hesperidin --- TMPRSS2 --- ACE2 --- D614G --- 501Y.v2 --- vitamin D deficiency --- 25(OH)D --- obesity --- CACFP --- early care and education --- food program --- food supplements --- immunity --- zinc --- lifestyle --- Poland --- clinical symptoms --- vitamin D status --- zinc status --- sunlight exposure --- prevention --- food hygiene --- bioactive compounds --- diet --- n/a --- diabetes mellitus --- oxidative stress --- kidney damage --- antioxidant

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This book presents the latest research published in the Special Issue Preventing the Adverse Effects of SARS-CoV-2 Infection and COVID-19 through Diet, Supplements, and Lifestyle in the journal Nutrients. In this book, we explore the role of supplements, lifestyle choices, and nutritional status on COVID-19 patient outcomes and risk reduction.

vitamin D --- COVID-19 --- SARS-CoV-2 --- severity --- mortality --- United Arab Emirates --- clinical trial --- saudi --- vitamin D insufficiency --- glycyrrhizin --- mineralocorticoid receptor --- toll like receptor 4 --- angiotensin converting enzyme --- aldosterone --- coronavirus --- exercise --- emotional eating --- pandemic --- quarantine --- questionnaire --- sedentariness --- New York --- hesperidin --- TMPRSS2 --- ACE2 --- D614G --- 501Y.v2 --- vitamin D deficiency --- 25(OH)D --- obesity --- CACFP --- early care and education --- food program --- food supplements --- immunity --- zinc --- lifestyle --- Poland --- clinical symptoms --- vitamin D status --- zinc status --- sunlight exposure --- prevention --- food hygiene --- bioactive compounds --- diet --- n/a --- diabetes mellitus --- oxidative stress --- kidney damage --- antioxidant

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Arterial hypertension affects about 1 billion people worldwide and it is the strongest modifiable risk factor for cardiovascular disease and related disability. Since the initial discovery of rare monogenic disorders with large effects, the role of genomics has evolved into large genome-wide association studies detecting common variants with a modest effect size. Similarly, pharmacogenomics has emerged as a new tool for understanding variability in drug response, to maximize efficacy and reduce toxicity. This book presents the most recent advances in the field of genetics and genomics of arterial hypertension and their potential impact on clinical management. The book is a useful tool for clinicians but also to the research community and those who want to be updated in the field.

Research & information: general --- Biology, life sciences --- atrial natriuretic peptide --- T2238C variant --- endothelial dysfunction --- smooth muscle cells contraction --- platelet aggregation --- epigenetics --- cardiovascular diseases --- renin --- low-renin --- hypertension --- mineralocorticoid receptor --- genetics --- aldosterone --- essential hypertension --- blood pressure --- genome-wide association studies --- exome microarray --- next-generation sequencing --- rare variants --- rare-variants association testing --- burden test --- sequence kernel association test --- hypokalemia --- low renin hypertension --- monogenic hypertension --- Liddle syndrome --- SCNN1A --- SCNN1B --- SCNN1G --- non-coding RNA --- micro RNA --- primary aldosteronism --- aldosterone-producing adenoma --- transcriptome profiing --- DNA methylation --- histone modifications --- vascular smooth muscle cells --- endothelial cells --- Kruppel-like factor 15 --- left ventricular hypertrophy --- cardiac hypertrophy --- heart failure --- genetics of left ventricular hypertrophy --- fibromuscular dysplasia --- non atherosclerotic vascular stenosis --- PHACTR1 --- genetic association --- cervical artery dissection --- spontaneous coronary arteries dissection --- CRY1 --- CRY2 --- HSD3B1 --- HSD3B2 --- cardio-tonic steroids --- endogenous ouabain --- adducin --- renal damage --- African American --- ARMC5 --- GRK4 --- CACNA1D --- endocrine hypertension